The cell cycle regulator 14-3-3σ opposes and reverses cancer metabolic reprogramming

نویسندگان

  • Liem Phan
  • Ping-Chieh Chou
  • Guermarie Velazquez-Torres
  • Ismael Samudio
  • Kenneth Parreno
  • Yaling Huang
  • Chieh Tseng
  • Thuy Vu
  • Chris Gully
  • Chun-Hui Su
  • Edward Wang
  • Jian Chen
  • Hyun-Ho Choi
  • Enrique Fuentes-Mattei
  • Ji-Hyun Shin
  • Christine Shiang
  • Brian Grabiner
  • Marzenna Blonska
  • Stephen Skerl
  • Yiping Shao
  • Dianna Cody
  • Jorge Delacerda
  • Charles Kingsley
  • Douglas Webb
  • Colin Carlock
  • Zhongguo Zhou
  • Yun-Chih Hsieh
  • Jaehyuk Lee
  • Andrew Elliott
  • Marc Ramirez
  • Jim Bankson
  • John Hazle
  • Yongxing Wang
  • Lei Li
  • Shaofan Weng
  • Nibal Rizk
  • Yu Ye Wen
  • Xin Lin
  • Hua Wang
  • Huamin Wang
  • Aijun Zhang
  • Xuefeng Xia
  • Yun Wu
  • Mouhammed Habra
  • Wei Yang
  • Lajos Pusztai
  • Sai-Ching Yeung
  • Mong-Hong Lee
چکیده

Extensive reprogramming of cellular energy metabolism is a hallmark of cancer. Despite its importance, the molecular mechanism controlling this tumour metabolic shift remains not fully understood. Here we show that 14-3-3σ regulates cancer metabolic reprogramming and protects cells from tumorigenic transformation. 14-3-3σ opposes tumour-promoting metabolic programmes by enhancing c-Myc poly-ubiquitination and subsequent degradation. 14-3-3σ demonstrates the suppressive impact on cancer glycolysis, glutaminolysis, mitochondrial biogenesis and other major metabolic processes of tumours. Importantly, 14-3-3σ expression levels predict overall and recurrence-free survival rates, tumour glucose uptake and metabolic gene expression in breast cancer patients. Thus, these results highlight that 14-3-3σ is an important regulator of tumour metabolism, and loss of 14-3-3σ expression is critical for cancer metabolic reprogramming. We anticipate that pharmacologically elevating the function of 14-3-3σ in tumours could be a promising direction for targeted anticancer metabolism therapy development in future.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2015